REBOUL CYRIL

Structure(s)

Laboratory or team

UPR 4278 LAPEC - Laboratory of Cardiovascular Experimental Physiology

Component and department

Biology Department
Institute of Agrosciences, Environment and Health

Research topics

Mitochondrial Redox Signaling and Its Impact on the Cardiac Stress Response

Publications

5 major publications : - Hyperglycemia triggers RyR2-dependent alterations of mitochondrial calcium homeostasis in response to cardiac ischemia-reperfusion: Key role of DRP1 activation. Dubois M, Boulghobra D, ... Reboul C. Redox Biol. 2024 Apr;70:103044. https://pubmed.ncbi.nlm.nih.gov/38266577/ - Exercise training may reduce fragmented mitochondria in the ischemic-reperfused heart through DRP1. Dubois M, ... Meyer G, Bornard I, Reboul C. J Gen Physiol. 2024 Dec 2;156(12):e202313485. doi: 10.1085/jgp.202313485. https://pubmed.ncbi.nlm.nih.gov/39508760/ - Increased protein S-nitrosylation in mitochondria: a key mechanism of exercise-induced cardioprotection. Boulghobra D, Dubois M, ... Reboul C. Basic Res Cardiol. 2021 Dec 23;116(1):66. https://pubmed.ncbi.nlm.nih.gov/34940922/ - Sinapine, but not sinapic acid, counteracts mitochondrial oxidative stress in cardiomyocytes. Boulghobra D, Reboul C*, Cazorla O*. Redox Biol. 2020 Jul;34:101554. doi: 10.1016/j.redox.2020.101554. Epub 2020 May 19 (*, co-corresponding authors) https://pubmed.ncbi.nlm.nih.gov/32464499/ - Exercise training protects the heart against ischemia-reperfusion injury: A central role for mitochondria? Boulghobra D, Coste F, Geny B, Reboul C. Free Radic Biol Med. 2020 May 20;152:395-410. doi: 10.1016/j.freeradbiomed.2020.04.005. Epub 2020 https://pubmed.ncbi.nlm.nih.gov/32294509/